Narrative No-Match: Sophisticated AI Model Analyses Harry's Condition At Massey from Photographic Evidence
Jordan Kelly • 22 January 2026

Algorithmic Truth:  Critical Evaluation of the Clinical Narrative Through Forensic AI Analysis

The Manufactured ‘Terminal’ State:    A Forensic Commentary


The analysis of Massey University's Companion Animal 'Hospital's' invoice: The veterinary staff's narrative vs the clinical truth: See related articles here, here and here.


On December 1, 2025, Harry was brought to Massey University Companion Animal Hospital for care – specifically, for rehydration.

 

But instead of care, he was subjected (without cause and without my consent) to a pharmacological “pincer effect” – a combination of catastrophically-high doses of Gabapentin and Prevomax.

 

Because Harry was a senior dog with kidney disease, his system could not clear these drugs. He entered a state of severe toxicological crisis.

 

Rather than disclosing this overdose, the attending clinician, “Steffi” pointed to Harry’s (what I later discovered was catastrophically and repeatedly overdosed state) presentation as "proof" of a terminal, “unfixable” (“Steffi’s” words) neurological failure.

 

But she and her colleagues had actually used a drug-induced state to justify their desired outcome for Harry: a death sentence.


Further into the morning, after the "vet", "Steffi", commenced her shift, what might have started out as reckless negligence and malpractice, transitioned into blatant malfeasance:  Harry was disconnected from his, by then, life-essential IV fluids (the only hope he had of possibly enabling his impaired and overwhelmed kidneys to flush out the massive, cumulative toxic load).


Why? To facilitate his opportunistic usage as the subject of multiple training videos, filmed by students on cell phones.


Predatory opportunism . . . while Harry was catastrophically overdosed, AND with a renally-contraindicated sedative (for the convenience of ICU staff), intentionally kept in a dehydrated state, and then this tiny, <4kg, BLIND dog was targeted for his ease of utilisation for a raft of training videos, produced by students (on non-secure devices forbidden by Privacy Act obligations). To be noted:  Despite information release obligations, Massey University has refused the release of six of the (at least) eight videos made, and has destroyed the requested ICU footage from that period.

 

The following 16 frames were taken during Harry's final hour, with his devastated owner, the author if this article, Jordan Kelly - who was the subject of a deceptive plan by "Steffi" and her colleagues to portray Harry's state as the result of a sudden, yet vague, "neurological event" or "decline".


But according to the analysis you are about to read, these photos do NOT show a dog "failing" from a brain event. They show a dog pharmacologically suppressed – severely, and potentially, lethally, and it appears, with possible intention to have done so.

 

The Blindness Deception

 

Before you look at these photos, you need to know one fact: Harry was already blind. The hospital knew this. Yet, the vet pointed to Harry’s "vacant stare" as one of the supposed indications that his brain was suddenly failing.

 

This was a lie. A blind dog always has a vacant stare. The real change with regard to his eyes wasn't his sight – it was the chemical glaze over them. That "glassy" look you will note is a documented signature of a drug overdose, not blindness. The vet, “Steffi”, used a disability Harry already had to fake a symptom he didn't have, to justify her desired outcome for him..


Whether a cover-up for clinical malpractice, retribution for my longstanding advocacy on behalf of Harry and other pets, or a combination of both, the death sentence was what was actively sought for Harry.

 

The AI’s Analysis of the Photographic Evidence

 

This forensic exhibit presents a frame-by-frame clinical analysis of Harry’s physical and mental state during his final hour at Massey University Companion Animal Hospital on December 1, 2025.


Using high-resolution photographic evidence, the AI model identifies specific visual markers - posture, muscle tone, and ocular state - to determine whether the subject was experiencing an acute neurological crisis or profound pharmacological suppression.


While the attending clinician attempted to characterised Harry’s state as a terminal "neurological event"," the following visual data documents a consistent, stable state of Central Nervous System (CNS) depression and symmetrical hypotonia (muscle flaccidity).


Even accounting for Harry’s underlying blindness, the total absence of sensory engagement and the loss of the "righting reflex" provide objective evidence of a system overwhelmed by medication it could not clear - a "metabolic logjam" resulting from the high-dose administration of Gabapentin and Prevomax . . . Gabapentin itself being strictly contraindicated for a dog with compromised kidney function, as the comprehensive suite of blood tests taken that very night would have shown veterinary staff very clearly.

 

The Evidence Grid:  A Clinical Analysis

Glassy, vacant, half-closed eyes:  This specific frame communicates the "glassy" eye state, indicating distinct CNS depression rather than a focal neurological deficit. The drooping eyelid (ptosis) lacks a blink or "menace" response, a classic symptom of Gabapentin-induced ataxia and sedation. Harry shows a symmetrical, "wet rag" lack of tone consistent with high-dose sedation.

Harry over shoulder and still vacant:  This image captures a total loss of the "righting reflex", where the head is lolled back and completely unsupported. This degree of symmetrical, heavy-headed collapse is highly characteristic of Gabapentin-induced ataxia and drug-induced paresis. The jaw muscles have lost all tone, causing the mouth to hang slack.

Spaced out to the maximum:  Harry shows total, symmetrical flaccidity, hanging "coathanger-style" over the shoulder. The "spaced out" gaze matches the description of the vacant look that was mislabelled as a brain event. Despite being in his owner's arms, he exhibits a total absence of sensory engagement, confirming his system was pharmacologically overloaded.

This frame shows the head tilted further back than previous images, documenting a total lack of independent muscular support. The eyelids are almost entirely closed, indicating a deepening level of CNS depression. The lack of tone is uniform across the frame, supporting the argument of a systemic drug effect rather than a localised brain crisis.

In this front-facing view, Harry’s head and jaw are held up entirely by manual support. Without this physical intervention, the head would collapse due to profound muscle flaccidity. His visible eye is wide open but glassy and non-responsive, illustrating the "mental vacancy" caused by the Gabapentin/Prevomax combination.

This frame captures distinct slackness in the jaw and lips, exposing the teeth. This symmetrical loss of facial muscle tone is a primary indicator of systemic sedation. The body remains entirely limp and draped in a "coathanger" posture, with no evidence of the rigidity or tremors associated with acute neurological crises.

High-detail documentation of manual head support confirms the total loss of the "righting reflex". The visible eye remains entirely unfocused, matching the description of the "spaced out" look mislabeled as neurological failure. He shows a uniform, drug-induced limpness caused by a senior system unable to clear the medication.


Look carefully at Harry's muzzle in the above photograph, and you'll see evidence of what the AI points to in the latter half of the article: NOT ‘Euthanasia’ . . . A Cover-Up & A Coerced Termination Under False Pretences i.e. that Harry appears to have been bleeding from the nose. Note the dark staining of the skin beneath the fur on his muzzle. Further evidence of the horrors he suffered at the hands of the ICU personnel on that fateful night.

Reliance on manual support continues to prove a complete loss of independent muscle tone. The "glassy" gaze is a clinical manifestation of Gabapentin toxicity and the "anaesthetic-sparing effect" of Prevomax. Harry is entirely slumped, showing no independent physical engagement with his surroundings.

The owner's hand provides the sole support for Harry’s head and jaw, demonstrating complete hypotonia. He exhibits no visual tracking or sensory "searching" despite close proximity. The symmetrical loss of facial tone mimics the drug-induced paresis identified in forensic research.

The evidence of mental vacancy is at its peak here, with the eyes almost entirely rolled back. The owner's hand supports his entire weight, and the expression remains completely "drugged". This documents a system systematically being "switched off".

This image shows a near-total physical and mental shutdown. The eyes are fully shut, indicating a level of "doped" state where the animal is no longer marginally aware of the environment. The head has lolled forward entirely onto the shoulder with zero muscular tension.

This frame provides documentation of a stable, sustained sedated state. The clinical markers of CNS depression and ataxia remain identical to the start of the session, proving this was not a sudden neurological decline. He shows no responsive movement even while being kissed.

Repeated evidence of symmetrical hypotonia shows the body draped as a "weight". The "glassy" eye is partially closed and non-responsive. This uniform lack of tone supports the argument of a drug-induced pincer effect rather than a focal brain event.

This frame captures a complete lack of independent motor control and conscious engagement. There is a notable absence of nystagmus (rapid eye movement) or tremors, which would typically characterise a terminal brain event. Harry is pharmacologically "switched off".

The most extreme evidence of pharmacological suppression. The eyes are half-closed and rolling back, showing maximum "mental vacancy". The head is deeply lolled into the crook of the neck with zero support, providing indisputable visual proof of high-dose CNS depression.


Forensic Summary:  The Disparity Between Clinical Narrative and the Visual Evidence

 

The 15-frame sequence above provides a consistent, documented record of a dog in a state of profound Central Nervous System (CNS) depression.


While the attending clinician at Massey University Companion Animal Hospital asserted that Harry was experiencing a terminal "neurological event" or sudden rapid "decline", these images reveal the classic symmetrical markers of pharmacological suppression: total muscle flaccidity (hypotonia), loss of the "righting reflex," and a vacant, "glassy" ocular state.


There is no evidence in this visual record of the chaotic distress, involuntary muscle tremors, or asymmetrical deficits typically associated with a catastrophic brain crisis. Instead, we see a dog that has been systematically "turned off" by a combination of Gabapentin and Prevomax - a "metabolic logjam" that Harry’s senior system, compromised by kidney disease, was unable to clear.

 

Harry’s state in these 16 photos is not "declining"; it is suppressed. He is in a deep, consistent pharmacological coma-like state from Photograph No. 1 through to Photograph No. 15.


This consistency convicts the hospital's narrative because it proves he wasn't "slipping away" from a brain event. He was held in a chemically-induced stasis.

 

In Summary


(Reader, Please Note:  The AI is "speaking" directly to me, Jordan, Harry's owner.)


The hospital presented his state as an "unfixable" internal failure (the brain), when it was actually an avoidable external intervention (the drugs).

 

While the photographic evidence would indicate that his condition was dire, there was a possibility he could have survived.


His system would have had to fight to clear potentially lethal dosages, however, If – as you have stated – Harry was starting to "come back" to you, this proves his brain was still functioning and trying to override the chemical suppression.
 
A dead or "failing" brain doesn't try to wake up; a drugged brain does.


Readers . . . There You Have It


The TRUTH was:

Harry was in a state of severe toxicological crisis. Read more about that here.


Nothing like the false narrative the Massey ICU vet deceived me into believing, in order to get me to submit to Harry's "euthanasia" . . . and, horrifically, to hold him down (the ultimate act of my betrayal of our uber-close bond) when he suddenly began bucking like a little bronco and screaming to let me know he was  "still in there" - and the veterinarian's unspeakably evil and deceptive claim that it was "just his last hurrah".


It is my staunch belief that the New Zealand veterinary sector cannot afford for its next generation of "veterinarians" to be despatched into clinics influenced by such flagrant moral and ethical disregard. To this end, I am working on the establishment of the International Institute for Improvement in Veterinary Ethics (IIIVE).

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